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In contrast, while the dose-response relationship between β-cell responsiveness to glucose (expressed as the slope of the linear relation between insulin secretion rate and the glucose concentration) and GLP-1 levels in circulation is partially impaired – a phenomenon referred as “GLP-1 resistance” (62) – a therapeutic infusion of GLP-1 at a pharmacological dose can restore β-cell insulin secretion responsiveness to glucose levels comparable to those observed in nondiabetic individuals (62)
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Poor sleep worsens insulin resistance and increases hunger hormones, counteracting the weight-loss benefit of the medication
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