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Our data indicate that exposure to semaglutide alone is sufficient to decrease proliferation of NEPC cells, which could be clinically significant for decreasing growth and increasing cell death in prostate cancers that are refractory to standard-of-care AR-targeted therapy (Figure 4B, D)
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Glimepiride differs from other agents in this class in that it is associated with equivalent metabolic control with weight-neutral effects on patients with T2DM
